Menopause : a comprehensive approach /

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Bibliographic Details
Imprint:	Cham : Springer, 2017.
Description:	1 online resource (337 pages)
Language:	English
Subject:	Menopause. Menopause Menopause.
Format:	E-Resource Book
URL for this record:	http://pi.lib.uchicago.edu/1001/cat/bib/11389009

Hidden Bibliographic Details
Other authors / contributors:	Cano, Antonio, 1954-
ISBN:	9783319593180 3319593188 9783319593173 331959317X
Digital file characteristics:	text file PDF
Notes:	""3.6.6.2 Shielding toÂ Reduce Radiation, Ovarian Transposition"" Includes bibliographical references. Print version record.
Summary:	This book provides an evidence-based approach to the universal experience of menopause. Its structured format clearly separates the biological basis from the clinical impact and quality of life, while also examining menopause within the context of healthy ageing in females. Accordingly, the book addresses factors including lifestyle, frailty, sarcopenia, and new ICT technologies. Written by respected experts in the field, the book offers a valuable guide for gynecologists and professionals devoted to women's healthcare and ageing quality of life, while also sharing revealing insights for non-professionals.
Other form:	Print version: Cano, Antonio. Menopause : A Comprehensive Approach. Cham : Springer International Publishing, ©2017 9783319593173
Standard no.:	10.1007/978-3-319-59318-0

Table of Contents:

Preface
Contents
Part I: Biological Background
1: Menopause, AÂ Stage inÂ theÂ Life ofÂ Women
References
2: Menopause: TheÂ Concepts andÂ theÂ Biological Background
2.1 Menopause andÂ theÂ Menopause Transition
2.2 Epidemiological Data
2.3 The Menstrual Cycle During Perimenopause
2.4 Variables Affecting Menopause Age
2.4.1 Genetics
2.4.2 Lifestyle
2.4.3 Other Factors
2.5 The Biological Basis ofÂ Menopause
2.5.1 Subclinical Phase
2.5.2 Cycle Irregularity
2.5.3 Postmenopause
References
3: Primary Ovarian Insufficiency3.1 Introduction
3.2 Pathogenesis ofÂ POI
3.2.1 Genetic Causes
3.2.1.1 Non-Syndromic POI
Ligands ofÂ theÂ TGF-Beta Family
BMP-15
GDF-9
Inhibin AÂ (INHA)
FMR1
G-Protein-Coupled Receptors
SF-1 (NR5A1)
Other Transcription Factors
FOXO3a
NOBOX
FIGLA
3.2.1.2 Syndromic POI
Turner Syndrome
Congenital Disorder ofÂ Glycosylation [Carbohydrate-Deficient Glycoprotein Syndrome (CDGs) Syndrome] andÂ Galactosemia
Pseudohypoparathyroidism Type 1a (PHP1A)
Autoimmune Polyglandular Syndrome Type 1Progressive External Ophthalmoplegia
Ovarioleukodystrophy
Ataxia Telangiectasia
Demirhan Syndrome
Blepharophimosis, Ptosis, Epicanthus Inversus Syndrome (BPES)
3.2.2 Autoimmune Causes ofÂ POI
3.2.3 Metabolic Disorders
3.2.4 Infectious Causes
3.2.5 Iatrogenic Causes
3.3 Epidemiology
3.4 Definition andÂ Clinical Presentation
3.4.1 Definition
3.4.2 Clinical Presentation
3.4.3 Personal andÂ Family History
3.4.4 Physical Examination
3.4.5 Initial Laboratory Assessment3.4.6 Further Investigation
3.4.6.1 Genetic
3.4.6.2 Autoimmune
Anti-ovarian Antibodies
Lymphocytic Oophoritis
Association withÂ Other Autoimmune Disorders
3.4.7 Risk Assessment forÂ Premature Ovarian Senescence
3.4.7.1 Pelvic Ultrasonography andÂ Ovarian Biopsy
3.4.7.2 AMH andÂ Inhibin B
3.5 Long-Term Consequences andÂ Follow-Up
3.5.1 Bone Health
3.5.2 Cardiovascular Disease
3.5.3 Psychological Aspects
3.5.4 Mortality
3.6 Management
3.6.1 Hormonal Therapy
3.6.1.1 Hormonal Supplementation inÂ Postpubertal POI Patients3.6.1.2 Puberty Induction
3.6.1.3 Nonhormonal Therapies
3.6.2 The Management ofÂ Patients withÂ Turner Syndrome
3.6.3 Surveillance ofÂ Cancer Survivors
3.6.4 Supportive Management
3.6.5 Reproductive Issues
3.6.5.1 Resumption ofÂ Ovarian Function andÂ Spontaneous Pregnancy Rate
3.6.5.2 Fertility, Pregnancy, Pregnancy Outcome
3.6.5.3 Obstetric Risks Associated withÂ POI
Turner Syndrome andÂ Pregnancy
3.6.6 Oncofertility
3.6.6.1 Fertility-Sparing Surgery

Menopause : a comprehensive approach /

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