Membrane trafficking in viral replication /

Saved in:
Bibliographic Details
Imprint:Berlin ; New York : Springer, c2005.
Description:1 online resource (vi, 259 p.) : ill. (some col.)
Language:English
Series:Current topics in microbiology and immunology, 0070-217X ; 285
Current topics in microbiology and immunology ; 285.
Subject:
Format: E-Resource Book
URL for this record:http://pi.lib.uchicago.edu/1001/cat/bib/8874502
Hidden Bibliographic Details
Other authors / contributors:Marsh, Mark.
ISBN:3540214305 (cased)
9783540214304 (cased)
3540267646 (electronic bk.)
9783540267645 (electronic bk.)
9786610304899
6610304890
Notes:Includes bibliographical references and index.
Other form:Print version: Membrane trafficking in viral replication. Berlin ; New York : Springer, c2005 3540214305
Description
Summary:Viruses are major pathogens in humans, and in the organisms with which we share this planet. The massive health and economic burden these agents impose has spurred a huge research effort to understand their most intimate details. One outcome of this effort has been the production, in many but certainly not all cases, of effective vaccines and therapies. - other consequence has been the realization that we can exploit viruses and put them to work on our behalf. Viruses are still seen to have the most - tential as vehicles for gene delivery and other therapeutic applications. However, their ability to exploit cellular functions to their own ends makes viruses not only highly effective pathogens but also exquisite experimental tools. Work with viruses underpins much of our current understanding of molecular cell biology and related fields. For membrane traffic in parti- lar, viruses have been crucial in providing insights into key cellular fu- tions and the molecular mechanisms underlying these events.
Physical Description:1 online resource (vi, 259 p.) : ill. (some col.)
Bibliography:Includes bibliographical references and index.
ISBN:3540214305
9783540214304
3540267646
9783540267645
9786610304899
6610304890
ISSN:0070-217X
;