Molecular basis of multiple sclerosis : the immune system /

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Bibliographic Details
Imprint:Heidelberg ; New York : Springer, c2010.
Description:1 online resource (viii, 307 p.) : ill. (some col.)
Language:English
Series:Results and problems in cell differentiation, 0080-1844 ; 51
Results and problems in cell differentiation ; 51.
Subject:
Format: E-Resource Book
URL for this record:http://pi.lib.uchicago.edu/1001/cat/bib/8895021
Hidden Bibliographic Details
Other authors / contributors:Martin, Roland (Physician)
Lutterotti, Andreas.
ISBN:9783642141539
3642141536
9783642141522
3642141528
Notes:Includes bibliographical references and index.
Description based on print version record.
Summary:Despite major efforts by the scientific community over the years, our understanding of the pathogenesis or the mechanisms of injury of multiple sclerosis is still limited. Consequently, the current strategies for treatment and management of patients are limited in their efficacy. The mechanisms of tissue protection and repair are probably even less understood. One reason for these limitations is the enormous complexity of the disease and every facet of its pathogenesis, the mechanisms of tissue injury, the diagnostic procedures and finally the efficacy of treatments and their side effects. The.
Other form:Print version: Molecular basis of multiple sclerosis. Heidelberg ; New York : Springer, c2010 9783642141522

MARC

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505 0 |a Molecular Basisof Multiple Sclerosis; Neuro-Immune Crosstalk in CNS Diseases; Role of NK Cells and Invariant NKT Cells in Multiple Sclerosis; Potential Triggers of MS; Prospects for Antigen-Specific Tolerance Based Therapies for the Treatment of Multiple Sclerosis; Immuno-Therapeutic Potential of Haematopoietic and Mesenchymal Stem Cell Transplantation in MS; Immune-Mediated CNS Damage; B Cells and Antibodies in MS; Immunological Basis for the Developmentof Tissue Inflammation and Organ-SpecificAutoimmunity in Animal Modelsof Multiple Sclerosis. 
520 |a Despite major efforts by the scientific community over the years, our understanding of the pathogenesis or the mechanisms of injury of multiple sclerosis is still limited. Consequently, the current strategies for treatment and management of patients are limited in their efficacy. The mechanisms of tissue protection and repair are probably even less understood. One reason for these limitations is the enormous complexity of the disease and every facet of its pathogenesis, the mechanisms of tissue injury, the diagnostic procedures and finally the efficacy of treatments and their side effects. The. 
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650 2 2 |a Molecular Biology  |x methods. 
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